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Summary Increasing evidence implicates altered mTORC1 signaling cascades in the pathophysiology of despair, suggesting that direct modulation of mTORC1 signaling might offer novel therapeutic prospective. In this particular concern from the JCI, Kato and colleagues reported that administration of NV-5138, a not long ago produced synthetic leucine analog, has a https://maynardi207zei1.wikifrontier.com/user

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